- S. aureus under stress by complement and coagulation
- Phagocytosis escape by a Staphylococcus aureus protein that connects complement and coagulation proteins at the bacterial surface
- The S. aureus polysaccharide capsule and Efb-dependent fibrinogen shield act in concert to protect against phagocytosis
- Complement activation by human antibodies against Staphylococcus aureus is governed by igG gexamer formation at the bacterial cell surface
- Antibody hexamerization improves Fc receptor-mediated phagocytossis of Staphylococcus aureus
- General discussion: Phagocyte modulation
