Contents:
- Staphylococcal complement inhibitor modulates phagocyte responses by dimerization of convertase
- Staphylococcal complement evasion by various convertase-blocking molecules
- Convertase inhibitory properties of the staphylococcal Extracellular complement binding protein
- Extracellular complement binding protein inhibits the interaction between C3d and complement receptor 2
- Immune evasion properties of C3 binding proteins
- Secreted immune evasion molecules contribute to aureus pathogenesis in vivo
